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dc.contributor.authorRosas Díaz, Tábata Victoria
dc.contributor.authorRodriguez-Bejarano, Eduardo 
dc.date.accessioned2019-11-15T13:28:39Z
dc.date.available2019-11-15T13:28:39Z
dc.date.created2019
dc.date.issued2019-11-15
dc.identifier.urihttps://hdl.handle.net/10630/18822
dc.description.abstractRNA interference (RNAi) in plants can move from cell to cell, allowing for systemic spread of an anti-viral immune response. How this cell-to-cell spread of silencing is regulated is currently unknown. Here, we describe that the C4 protein from Tomato yellow leaf curl virus can inhibit the intercellular spread of RNAi. Using this viral protein as a probe, we have identified the receptor-like kinase (RLK) BARELY ANY MERISTEM 1 (BAM1) as a positive regulator of the cell-to-cell movement of RNAi, and determined that BAM1 and its closest homologue, BAM2, play a redundant role in this process. C4 interacts with the intracellular domain of BAM1 and BAM2 at the plasma membrane and plasmodesmata, the cytoplasmic connections between plant cells, interfering with the function of these RLKs in the cell-to-cell spread of RNAi. Our results identify BAM1 as an element required for the cell-to-cell spread of RNAi and highlight that signalling components have been co-opted to play multiple functions in plants.en_US
dc.description.sponsorshipUniversidad de Málaga. Campus de Excelencia Internacional Andalucía Tech.en_US
dc.language.isoengen_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.subjectCélulas vegetalesen_US
dc.subject.otherGeminivirusen_US
dc.subject.otherSilencingen_US
dc.subject.otherC4en_US
dc.titleA viral effector suppresses cell-to-cell spread of silencing by targeting two plasmodesmal receptor-like kinasesen_US
dc.typeinfo:eu-repo/semantics/conferenceObjecten_US
dc.centroFacultad de Cienciasen_US
dc.relation.eventtitleInternational Advances in Plant Virologyen_US
dc.relation.eventplaceRoma-Italiaen_US
dc.relation.eventdate29 de octubre 2019en_US


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