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Tumor immune escape by autotaxin: keeping eosinophils at bay

dc.contributor.authorMatas-Rico, Elisa
dc.contributor.authorMoolenaar, Wouter H
dc.date.accessioned2024-09-25T12:39:48Z
dc.date.available2024-09-25T12:39:48Z
dc.date.issued2024-04
dc.identifier.citationElisa Matas-Rico, Wouter H. Moolenaar, Tumor immune escape by autotaxin: keeping eosinophils at bay, Trends in Cancer, Volume 10, Issue 4, 2024, Pages 283-285, ISSN 2405-8033, https://doi.org/10.1016/j.trecan.2024.03.002. (https://www.sciencedirect.com/science/article/pii/S2405803324000517)es_ES
dc.identifier.urihttps://hdl.handle.net/10630/33278
dc.description.abstractSecreted autotaxin (ATX) promotes tumor progression by producing the pleiotropic lipid mediator lysophosphatidic acid (LPA). In a recent Nature Cancer paper, Bhattacharyya et al. show that ATX/LPA signaling suppresses CCL11-driven infiltration of eosinophils into the pancreatic tumor microenvironment to facilitate tumor progression, thus revealing a new ATX-mediated immune escape mechanism and highlighting the antitumor potential of eosinophilses_ES
dc.language.isoenges_ES
dc.publisherElsevieres_ES
dc.subjectCánceres_ES
dc.subject.otherImmunity, Cancer, autotaxin, lysophosphatidic acid, tumor microenvironmentes_ES
dc.subject.otherImmunityes_ES
dc.subject.otherTumor microenvironmentes_ES
dc.subject.otherLysophosphatidic acides_ES
dc.subject.otherAutotaxines_ES
dc.titleTumor immune escape by autotaxin: keeping eosinophils at bayes_ES
dc.typejournal articlees_ES
dc.centroFacultad de Cienciases_ES
dc.identifier.doi10.1016/j.trecan.2024.03.002
dc.type.hasVersionVoRes_ES
dc.departamentoBiología Celular, Genética y Fisiología
dc.rights.accessRightsopen accesses_ES


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